
Research & Development
Promoting discovery through advanced scientific research
Centro Ciencia & Vida is one of Chile’s leading hubs for science, bringing together over 120 researchers united by the goal of expanding knowledge and driving innovation. Their work spans three main fields—Biomedicine, Computational Biology, and Microbiology—which are developed in five collaborative clusters. By organizing research in this way, CCV nurtures both scientific excellence and collaboration at the service of people.
Laboratories
At Centro Ciencia & Vida, laboratory teams investigate biomedicine, microbiology, and computational biology through end-to-end workflows.
Standardized SOPs, quality controls, and technology transfer pipelines ensure reproducible science that translates into diagnostics, therapies, and biotech innovations.
Publications
Explore peer-reviewed publications across our five research clusters, reflecting international collaboration and translational impact. Indexed outputs have met or exceeded annual targets, and citation rates have risen above projections.
Together, these publications showcase the synergy across clusters and the CCV's progression from foundational discovery to application.
Clusters
The five clusters focus on high-impact diseases (autoimmunity, infections, cancer, and neurodegenerative disorders) and strategic areas such as pandemic preparedness and biomining.
This synergistic structure translates world-class discoveries into technologies and advanced human capital, positioning Chile as a competitive hub for biomedical innovation and talent.
Viruses, Microbes and Infections
Molecular basis of infection by human viral pathogens.
Role of chromatin modification, histone-like proteins and membrane fusion proteins in viruses and mobile genetic elements.
Role of gut microbiota in the inflammatory behavior of autoreactive lymphocytes and in intestinal homeostasis and perturbation.
Cell Signaling and Neuropathology
Stress and synaptic homeostasis.
Synaptic mitochondrial dysfunction, accumulation of abnormal proteins and protein degradation in the mitochondria during neurodegeneration and aging.
Antibody-driven brain function.
Adaptive immunoreceptors and Parkinson disease.
Fibrosis in skeletal muscle diseases.















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